تلخیص
The present research was conducted to estimate the gene action for inheritance of resistant to CLCV disease. Adequacy of additive and dominance model was conducted, both scaling test designated that hypothesis was fit for genetic analysis. Significant of both additive (D) and dominance parameters (H1 and H2 ) endorsed the presence of additive and dominance genes for inheritance of resistant to CLCV. Additive (D) effect was more prominent than dominance (H1 and H2 ) suggested additive genes in legacy of resistant in both generations. Uneven values between H1 and H2 is sign of unequal dispersal of recessive and dominance genes. Values of H2 /4H1, was less than (0.25), recommended allelic frequency of positive and negative effects gene was asymmetrical. F parameter, √ √ and √ √ values were also proved equal proportion with relative frequency of dominant and recessive alleles in parents, h2 value directed the existence of dominance effects due to heterozygous loci. Genetic ratio h 2 /H2 signalized that at least one genetic group is involved in controlling to resistance of CLCV disease in F1 and F2 generation. High narrow sense heritability indicated, selection in early segregating generations, because of traits of genotypes heritage to its offspring. Resistant offspring in F1 and due to segregation F2 population ratio was observed as 3:1 (resistant plants: diseased plants) among Mac-7 population, which legitimated the single dominant gene proved monogenic dominance nature of CLCV disease resistant in cotton. The Wr-Vr graph also shown Mac-7 carried maximum dominant genes and USD16-3058 contained maximum recessive genes. While, rest of the parents had different constitution of dominant and recessive genes. The high values of PCV and GCV proved that resistant of CLCV did not affected due to environment. High board sense heritability coupled with high genetic advance, suggested role of additive genes. Therefore, single plant selection could be fruitful in early segregating generation.